Chapter 12 - FAP CNS
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FAP CNS 01 – CAPS Reflects the end of an Increasing Disinhibition/Decreasing Afferent
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FAP CNS 02 – Pain is a Modifiable Experience
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FAP CNS 03 – Visceral Hypersensitivity in IBS but not in CAPS
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FAP CNS 04 – Catastrophizing is a Process in Which an Individual Amplifies Their Pain
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FAP CNS 05 – Catastrophizing-When Feelings Become Facts
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FAP CNS 06 – Symptom-Related Behaviors in CAPS
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FAP CNS 07 – Psychosocial Assessment in CAPS
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FAP CNS 08 – Carnett’s Test
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FAP CNS 09 – Constant or Frequency Recurring Abdominal Pain
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FAP CNS 10 – Multicomponent Approach to Centrally Mediated Abdominal Pain
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FAP CNS 11 – Important Steps that Patients and Clinicians can do to Establish
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FAP CNS 12a – Effect of Imipramine vs. Placebo on Cognitive Function in TBI
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FAP CNS 12b – Effect of Imipramine vs. Placebo on Hippocampal Cell Proliferation
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FAP CNS 13 – Relationship of Chronic Opioid Use, OBD, OIC, and NBS
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FAP CNS 14 – Bimodal (Excitatory and Inhibitory) Opioid Modulation System
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FAP CNS 15 – Bimodal Opioid Modulation in Dorsal Horn
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FAP CNS 16 – Pain Response (Abdominal Contractions) by Day to Morphine vs. Vehicle
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FAP CNS 17 – Risk of Refilling Narcotics by Days Since Detoxification
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FAP CNS 18 – Abdominal Pain Scores Pre and Post Detoxification
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FAP CNS Table 1 – Factors Affecting Patient-Physician Relationships in CAPS
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FAP CNS Table 2 – Antidepressant Treatment
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FAP CNS Table 3 – Antidepressant Receptor Site Effects
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FAP CNS Table 4 – Factors Associated with Detoxification Failure or Recidivism
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FAP CNS Table 5 – Treatment Approach-Opioid Detoxification
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